If you reside in a house or apartment or simply want to keep your home free of electromagnetic fields There are plenty of ways you can reduce exposure. http://cavecut28.xtgem.com/__xt_blog/__xtblog_entry/__xtblog_entry/33689227-information-about-emf-blocking-radiation?__xtblog_block_id=1#xt_blog of the most effective is to reduce your electronic device use. It is also possible to use EMF blocker paint to block EMF radiation from entering your home. Another method to protect your home from EMF radiation would be to use a RF shielding canopy. This is a cloth of net that contains EMF shielding. It is utilized to stop EMFs from entering a space. Another option is to get your home equipped with an enclosure that is conductive. These devices are called Faraday cages.
Numerous studies have demonstrated studies have shown that the nonionizing EMF has anti-proliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed result from the deregulation the cancer stem cell. https://dugan-ahmed.mdwrite.net/emf-block-paint-and-even-emf-shielding-1681434137 could explain the long-term effects observed in some patients with advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not clear.
Effects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumours were classified into three groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to that used in humans. The third group was exposed RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumours was evaluated against that of RCF. The results showed that the tumours treated with HCCMF had significant shrinkage. However, https://fnote.me/notes/yM31Kq treated with RCF showed no evidence of tumour shrinkage.
The reason for cancer-specific AM RF EMF might be due to the fact that tumor cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is mediated through CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H may have broader implications for treatment and diagnosis of various cancers.
The tumors in the controls were never exposed RF EMF, and were fed a standard mouse diet. The tumors of the HCCMF group were infected with Huh7 cells after they were between five and seven weeks old. The tumours were then euthanized when they showed excessive burden.
The tumours in the three groups also showed different growth curves. The HCCMF-treated tumors showed a significant decrease in tumour size after 8 weeks. However, tumors that were treated using RCF did not show any shrinkage. The difference was highly significant. The tumors treated with RCF showed necrosis, which is common in tumors that have been exposed to RCF. It is possible that this necrosis was caused by the lack of oxygen in the more invasive tumours.
In summary, the results show that AM RF EMF exhibits anti-cancer properties in vitro as well as in vivo. Several studies have shown it is true that AM RF EMF produces measurable shrinkage of tumors within HCC patients. There is a possibility that AM RF EMF produces these effects through CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained impact on the development of HCC tumours in the vivo.